Each of these receptors activates G protein-coupled inwardly rectifying potassium (GIRK) channels. have shown that co-activation using saturating concentrations of agonists results in occlusion of the GIRK The results indicate that a low tonic level of G βγ results in facilitation of GIRK current and a high The results suggest that the cooperative interaction between G βγ subunits and GIRK channels determines Activation of either D2 or GABAB receptors also acutely desensitized the GIRK channel current induced

II-induced calcium signaling in striatal neurons Receptor-dependent influence of R7 RGS proteins on neuronal GIRK

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