Deficiency of β-arrestin2 alleviates apoptosis through GRP78-ATF6-CHOP signaling pathway in primary Sjögren's In vivo, we found that inhibition of GRP78-ATF6-CHOP apoptosis signaling improved ESS symptoms, and the salivary gland indices, and improved tissue integrity in the ESS model by downregulating GRP78-ATF6-CHOP In addition, β-arrestin2 depletion downregulated GRP78-ATF6-CHOP apoptosis signaling to alleviate cell of pSS and that β-arrestin2 encourages inflammation-induced epithelial apoptosis through GRP78-ATF6-CHOP

References: Jin, B. K., Odongo, S., Radwanska, M., & Magez, S. (2023). G., Choi, H. J., Fung, J. J., Pardon, E., Casarosa, P., Chae, P. S., Devree, B. T., Rosenbaum, D.

Structure-guided design of a peripherally restricted chemogenetic system Hye Jin Kang , Brian E Krumm

Jacob Lee and Jin Cho, co-founders of GeneMode, on the ten billion dollars American labs waste every In this conversation, Jacob Lee and Jin Cho — co-founders of GeneMode — describe what they saw when they Jin Cho is the CTO and co-founder of GeneMode. Jacob Lee and Jin Choe met in ninth grade in English class and have been friends since. GPCR Ecosystem Jin Choe on LinkedIn Jin Choe on Dr. GPCR Ecosystem Unlock the Full Dr.

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