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Deficiency of β-arrestin2 alleviates apoptosis through GRP78-ATF6-CHOP signaling pathway in primary Sjögren's In vivo, we found that inhibition of GRP78-ATF6-CHOP apoptosis signaling improved ESS symptoms, and the salivary gland indices, and improved tissue integrity in the ESS model by downregulating GRP78-ATF6-CHOP In addition, β-arrestin2 depletion downregulated GRP78-ATF6-CHOP apoptosis signaling to alleviate cell of pSS and that β-arrestin2 encourages inflammation-induced epithelial apoptosis through GRP78-ATF6-CHOP

References: Jin, B. K., Odongo, S., Radwanska, M., & Magez, S. (2023). G., Choi, H. J., Fung, J. J., Pardon, E., Casarosa, P., Chae, P. S., Devree, B. T., Rosenbaum, D.

Structure-guided design of a peripherally restricted chemogenetic system Hye Jin Kang , Brian E Krumm

Hormone Receptors and Hearing and Balance Receptors Abstract Only Available for AGPCR24 Attendees About Jin-Peng Jin-Peng Sun on the web Google Scholar LinkedIn < Previous Session Next Session >

GPCR Podcast << Back to podcast list Chloe Hicks About Chloe Hicks Chloe Hicks will graduate from Duke Chloe Hicks on the web ORCID LinkedIn Dr. GPCR Enjoying the Dr. GPCR Podcast? Leave a Review.

Jin-Peng Sun’s Lab since I graduated from Shandong University in 2012, and worked under the guidance